This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowReprints and Permissions
Right arrow Copyright Information
Right arrow Books from ASM Press
Right arrow MicrobeWorld
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Burian, K.
Right arrow Articles by Gonczol, E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Burian, K.
Right arrow Articles by Gonczol, E.

 Previous Article  |  Next Article 

Clinical and Diagnostic Laboratory Immunology, November 2001, p. 1263-1266, Vol. 8, No. 6
1071-412X/01/$04.00+0   DOI: 10.1128/CDLI.8.6.1263-1266.2001
Copyright © 2001, American Society for Microbiology. All rights reserved.

Chlamydia pneumoniae Exacerbates Aortic Inflammatory Foci Caused by Murine Cytomegalovirus Infection in Normocholesterolemic Mice

Katalin Burian,1 Klara Berencsi,2 Valeria Endresz,1 Zsofia Gyulai,1 Tibor Valyi-Nagy,3 Istvan Valyi-Nagy,4 Marta Bakay,1 Yuemei Geng,2 Dezso Virok,1 Laszlo Kari,2 Rozalia Hajnal-Papp,5 Giorgio Trinchieri,2 and Eva Gonczol1,2,*

Department of Medical Microbiology1 and Department of Pathology,5 University of Szeged, Szeged, and First Department of Internal Medicine, Semmelweis University, Budapest,4 Hungary; The Wistar Institute, Philadelphia, Pennsylvania2; and Department of Pathology, Vanderbilt University, Nashville, Tennessee3

Received 26 February 2001/Returned for modification 9 May 2001/Accepted 18 July 2001

Inflammatory foci induced by murine cytomegalovirus infection in normocholesterolemic mice were present temporarily in the aortic wall, but some of these foci developed into advanced lesions that persisted late after infection. The early foci induced by virus infection were significantly exacerbated following a single inoculation with Chlamydia pneumoniae.

* Corresponding author. Mailing address: The Wistar Institute, 3601 Spruce St., Philadelphia, PA 19104. Phone: (215) 898-3887. Fax: (215) 898-3868. E-mail: Gonczol{at}wistar.upenn.edu.

Clinical and Diagnostic Laboratory Immunology, November 2001, p. 1263-1266, Vol. 8, No. 6
1071-412X/01/$04.00+0   DOI: 10.1128/CDLI.8.6.1263-1266.2001
Copyright © 2001, American Society for Microbiology. All rights reserved.




Copyright © 2009 by the American Society for Microbiology.   For an alternate route to Journals.ASM.org, visit: http://intl-journals.asm.org | More Info»